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In CPT, the induced local seismicity density is proportional to the pressure rate, limited by the Kaiser Effect, and apparently un-related to the tectonic background seismicity.
In CPT II and VLCAD deficiencies, fatty acid oxidation (FAO) is normal at rest, but residual enzyme activity is insufficient to increase FAO during exercise.
Therefore, it is necessary to obtain transcriptome data and screen candidate transcripts involved in CPT biosynthesis to further understand the CPT biosynthetic pathway.
The obtained results demonstrate that the root is not involved in CPT biosynthesis, although it is a site of CPT accumulation.
According to EST annotation, catalytic features prediction, and expression analysis, novel putative transcripts involved in CPT biosynthesis and transport were discovered in C. acuminata.
We will analyze the data problem in CPT learning and propose a retrieval algorithm.
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Therefore, we speculate that transcripts of CaG10H, CaSCS, and CaSTR were most likely to be involved in CPT biosynthesis.
We identify a sequence insertion found in CPT-2 that mediates membrane localization.
The results also demonstrated an increased linker flexibility of the CPT resistant Top1, providing an additional explanation for the resistance mechanism found in CPT-producing plants.
Finally, how CPT-producing organisms resist their own toxic metabolite and how this knowledge may potentially be of use in CPT-resistance management is covered.
However, deficiency in SAE2 caused only a very modest increase in CPT-sensitivity (Fig. 5B) or ETP-sensitivity (Fig. 5C).
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