Suggestions(1)
Exact(6)
RSU i responds immediately to v1, if the set is always available in the storage; otherwise, if the set is not existent or has been expired, RSU i will forward the request to its server on the Internet, denoted as Root.
The convergence order is computed by the following formula: order ≃ log ( E i / E i + 1 ) log ( h i / h i + 1 ), where i responds to the spatial partition, and E i denotes the for the state, costate and control approximation.
Under these assumptions, the probability p ij that sample i responds to drug j is given by (6) p ij = 1 2 erfc log IC 50 ij − mean log IC 50 j − log h 2 σ where erfc(x) is the complementary error function.
Acquired and congenital cases differ in that ACD (i) responds partially to potassium replacement, (ii) may have small quantities of chloride in the urine rather than zero often reported in CCD [ 11], (iii) appears to be associated with bowel resections and ostomies, and (iv) can be transient and reversible with surgical correction of the bowel abnormality.
Using this notation we obtain the response by-marker approximation (5) p ¯ ij = q j, s j X i In short, the probability that a given patient i responds to a given drug j is approximated by the estimated fraction of patients that responds to that drug within the group of patients having the same status as patient i for the markers assigned to drug j.
Also like DPB, BLS I responds favorably to erythromycin therapy by showing a resolution of symptoms.
Similar(54)
These results further indicate that RIG-I responds to poly-I/C as well as to pppRNA.
RIG-I responds to in vitro-transcribed dsRNA, vesicular stomatitis virus (VSV), Newcastle disease virus (NDV), and influenza virus in mice.
Previously, it has been demonstrated that RIG-I responds to infection by SV, VSV, NDV, and influenza virus, whereas MDA5 is involved in IFN signaling triggered by poly (I∶C) transfected into the cytoplasm [3], [19].We further determined whether REUL played a role in the cellular antiviral response mediated by RIG-I.
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