Sentence examples for I interaction from inspiring English sources

Two main micromechanical effects are conceivable in this context, namely: (i) interaction of γRN with microcracks from the matrix (crack blunting or arresting); and (ii) deformation-induced phase transformation of γRN to martensite (TRIP effect).

Then the proposed numerical model is used to obtain flow results for the following free surface flow cases: (i) interaction between two opposite solitary waves, (ii) seiche phenomenon in a rectangular reservoir, and (iii) solitary wave through a submerged rectangular structure in a viscous fluid.

Compared to Fe3O4 NPs, MCPs have lower M s which is attributed to the presence of nonmagnetic ZnO shell on the surface of Fe3O4 through (i) interaction of surface atoms of magnetite with nonmagnetic layer of ZnO [20] and (ii) surface defects and strain in nanocomposite [21].

Three major interactions, i) interaction between genes for the same trait, ii) genes for different traits, and iii) interactions of genes with environments and genetic background restricting the use of QTLs in introgression programs (Kumar et al. 2014; Wang et al. 2012; Xue et al. 2009; Almeida et al. 2013; Elangovan et al. 2008; Cuthbert et al. 2008; Heidari et al. 2011; Bennett et al. 2012).

Our data suggest that the TRIM5αrh-mediated late restriction involves at least two distinct activities: (i) interaction with HIV-1 Gag polyprotein through the N-terminal, RING and b-box 2 regions of a TRIM5αrh monomer, and (ii) an effector function(s) that depends upon the coiled-coil and linker 2 domains of TRIM5αrh [19].

This paradoxical effect could be caused by: (i) interaction of the donor apoptotic cells with direct pathway T cells or, more likely, (ii) down-regulation of the indirect CD4 T cell help (through T cell deletion or generation of Treg) required for maturation of direct T cells into effector cells [76], [77].

In [15, 16], the authors explored the effect of HTLV-I interaction through a system of delay differential equations (DDEs).

The E-to-I interaction in PING is an example of an excitatory signal terminated by a feedback signal from the target: The E-cells stop firing when the I-cells respond, and thereby they shut them off.

In order to obtain mechanistic insights into the RIG-I interaction with RNA, we searched the pdb for possible structural homologues.

Furthermore, EGCG dose-dependently inhibited the ATPase activity of recombinant RIG-I but did not compete with RIG-I interaction with RNA or with ATP.

EK 293T cells were transiently co-transfected with 1 µg of Flag-tagged MAVS or V5-tagged 2CARD vectors (for tandem 2CARD/MAVS interaction analysis) or 3 µg of Flag-tagged 2CARD and biotin-conjugated RIG-I vectors (for tandem 2CARD/RIG-I interaction).