Sentence examples for I efflux from inspiring English sources

Primary functional classes of the regulated genes were (i) efflux pumps, (ii) stress response, (iii) fatty acid, lipid, and derivatives biosynthesis, and (iv) hexose transport, ergosterol biosynthesis, and thiamine biosynthesis (Table  1).

To measure the I efflux, cells were cultured for 1 h with 18.5 kBq of NaI, then the old medium was decanted and cells were cultured for 0, 5, 10, 15, and 20 min. All experiments were repeated 3 times.

CFTR chloride channel activity was assayed by measuring iodide (I) efflux from transfected HeLa cells as described previously (Marivingt-Mounir et al, 2004).

In addition, W619 has a copF gene putatively encoding a P1-type ATPase, which is located in the inner membrane and involved in Cu(I) efflux from the cytoplasm to the periplasm (Rensing et al., 2000; Mergeay et al., 2003;).

Although much of the structural work in bacterial systems has focused on the molecular details of Cu(I) transfer from the chaperone to the CopAMBD, recent evidence, at least for Archaeoglobus fulgidus (Af) CopA, suggests that this transfer is not on pathway for Cu(I) efflux, but instead plays a regulatory role in allosteric activation of CopA.

Resistance proteins include cytoplasmic copper chelators like bacterial metallothionein (MymT) in M. tuberculosis, copper chaperones that operate in both the periplasm and the cytoplasm, multicopper oxidases (MCOs) that oxidize Cu(I) to less toxic Cu II), and Cu(I) efflux pumps and outer membrane-localized channels that move copper across membranes.

Recent highlights of copper chemistry at the host microbial pathogen interface include the first high resolution structures and functional characterization of a Cu(I -effluxing P1B-ATPase, a new class of bacterI -effluxinghaperone, a fungal Cu-only superoxide dismutase SOD5, anewthe disclassy of a small molecule Cu-bacterial mimeticopper

The rate of "leakiness," or permeability to bacteria (i.e., efflux of bacteria), of the intestinal epithelial layer is treated as a variable.

In principle, there are also pathways for glucosamine metabolism, which could result increased glycolytic flux [37], [38]; however, glucosamine had no effect on either glucose oxidation or glucose-derived lactate efflux, i.e., glycolysis (Figure 3).

This latter observation indicates that cellular compensation mechanisms, such as reduced apical influx, increased basolateral efflux (i.e. expression of znt1), and cytosolic chelating capacity.

Moreover, anomalies increase in root-forming NAA-cultured hypocotyl segments when an inhibitor of auxin efflux, i.e. 1-naphthylphthalamic acid, is also applied (Pernisová et al., 2009).