Sentence examples for I deletion from inspiring English sources

(i) Deletion 8 Increasing learners' ability for complications Transitioning to advanced problems by making the problems more complicated.

These modifications involved (i) deletion of the gene encoding the repressor LtbR to increase expression of leuBCD, (ii) deletion of the gene encoding the transcriptional regulator IolR to increase glucose uptake, (iii) reduction of citrate synthase activity to increase precursor supply, and (iv) introduction of a gene encoding a feedback-resistant acetohydroxyacid synthase.

We modified the wild-type Tth pol by (i) deletion of the N-terminal 5′ to 3′ exonuclease domain, (ii) fusion with the DNA-binding protein Sso7d, (iii) introduction of four known effective point mutations from other DNA polymerase mutants, and (iv) codon optimization to reduce the GC content.

(i) Deletion of genes coding for protective immunogens in BCG and/or over attenuation of BCG, due to the loss of genetic Regions of Differences (RD) 7, 8 could explain the weak efficiency of BCG.

Thus, maternally inherited copy changes were considered as of unclear clinical significance and noted as: (i) deletion in 7p22.1 for ambiguous genitalia; (ii) duplications in 4q35.2 and 5p15.31 for hypospadias; (iii) duplication in 5p15.2 and in the androgen receptor insensitivity region, Xq12 for cryptorchidism (Table 5).

This hypothesis is based on the following observations: (i) deletion or a kinase-dead mutation in IME2 results in a delayed entry into meiotic S and nuclear division [21], [27], (ii) in cells expressing the ime2-3SA allele meiotic S is induced prematurely (Figure 7A), and (iii) Ime2 is essential for the transcription of NDT80 [36].

De novo occurrences were noted for: (i) deletions in 1p36.33, 9p23p24 and 19q12-q13.11 for probands presenting with a referring diagnosis of ambiguous genitalia; (ii) duplications in 10p14 and Xq28 for cryptorchid children; (iii) and deletions in 12p13.31-p13.2 and 16p11.2 for patients with hypospadias (Table 6).

Furthermore, EGCG did not inhibit the constitutive activities of RIG-I deletion mutants R-C and R-CH (Figure 4A).

Similarly, we made RIG-I deletion mutants and determined which domain is required for interaction with REUL.

Barber et al. have predicted that RIG-I deletion might underlie the sensitivity of chickens to avian influenza [ 15].

Also, Barber et al. and Huang et al. proposed that duRIG-I deletion might underlie the sensitivity of chicken to avian influenza [ 15, 16].