Exact(32)
For fixed values of different variables, it is clear that increase in R i causes an increase in (bar{I}_{i}).
During steam curing, thermal extension of the strand at time t i causes local slip and bond stress at location x between the strand and the surrounding concrete [s(x) and τ(x) in Fig. 1, respectively].
For example, considering the GC-2 2,7) GC-2 2,7m algorithmN 0 > 7.5 dB, it is very probandE that the bit inversion resulting from the addition of test patterns b i causes errors in the sequence y.
If μ i < 1, a one per cent increase in the before-tax real wage w i causes a less than one per cent increase in the after-tax real wage ω E i, indicating that the income tax is progressive.
This is given as μ f = E density of active dominant interferers for RX 0 = Pr TX i placed at ( x, y ) · Pr ( TX i − RX i activated | ( x, y ) ) · Pr ( TX i causes error at RX 0 | TX i active at ( x, y ) ) (24) = λ 1 − e − h ii R − α r α β t 1 − e − h ii R − α r α β r e − h 00 R − α r α β. (25).
An increase in [Ca2+] i causes an increase in the ratio [ 34].
Similar(28)
Over-expression of IGF-I causes profound alterations in the vascularisation of the mouse eye [48] but to our knowledge there are no reports of eye defects associated with IGF-I deficiency.
Accordingly, the Igf1−/− mouse shows poor growth rates, high mortality, profound sensorineural deafness and late postnatal morphological alterations in the cochlea [16] We have shown previously that the absence of IGF-I causes poor myelination and delayed maturation of auditory neurones that suffer apoptosis during the early postnatal mouse development P5-P20 [18], [18].
These data, together with the reported morphological alterations and the profound sensorineural deafness that the deficit in IGF-I causes in mice and humans, prompted us to further study the molecular mechanisms underlying IGF-I activity in the developing mouse cochlea.
PDE-I causes the activation of cyclic adenosine monophosphate (cAMP) and protein kinase A (PKA), resulting in altered calcium handling by sarcoplasmic reticulum (SR) [ 11].
BinTX-I causes mouse hind-paw edema, as well as myonecrosis and partial neuromuscular blockade in chick biventer cervicis preparations [ 98].
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