Sentence examples for Extended viability from inspiring English sources

The extended viability of anti-miR-466h CHO cells was the result of delayed Caspase-3/7 activation by more than 35 h, and the increased levels of its anti-apoptotic gene targets (smo, stat5a, dad1, birc6, and bcl2l2) to between 2.1- and 12.5-fold compared with the negative control CHO in apoptotic conditions.

They confirmed that incubation at 5°C extended viability of cells of C. jejuni, and incubation at 10°C significantly prolonged the viability of Campylobacter.

Importantly, CMS-induced cells demonstrated extended viability under metabolic stress, as the medium depleted with glucose after 5 7 days, with 90% cell death being delayed by at least a week compared with parental cells.

However, the mild embryonic vascular phenotypes combined with the extended viability of Nrp1 Y297A/Y297A relative to full and endothelial-specific NRP1 knockout mice clearly demonstrates for the first time that VEGF binding to NRP1 is not essential for embryonic angiogenesis and, therefore, that NRP1 functions in embryonic ECs predominantly in a VEGF-independent role.

These engineered cells reached higher maximum viable cell density and extended viability compared with negative control and parental CHO cells in batch cell cultures which resulted in the 53.8% and 41.6% increase of integral viable cells.

A natural tolerance of various environmental stresses is typically supported by various cytoprotective mechanisms that protect macromolecules and promote extended viability.

Here, we report on a novel STAT3-mediated pathway that characterizes CLL B cells-extended viability and oxidative stress control.

López de Lacey, López-Caballero, Gómez-Estaca, Gómez-Guillén, and Montero (2012) reported that Lactobacillus acidophilus and Bifidobacterium bifidum entrapped in gelatine based coatings stored for 10 days at 2 °C showed extended shelf life and prolonged viability.

Results have shown that sandwich cultures with matrix proteins can lead to prolonged hepatocyte viability (Dunn et al. 1991), extended CYP activity (Kern et al. 1997) and increased cell polarization toward more advanced bile canaliculi networks (LeCluyse et al. 1994).

Cell viability in extended culture was maintained as analyzed by expression of Cell Tracker green and negligible ethidium homodimer-1 (a marker of cell necrosis) staining.

Consistent with the incompatibility of extended PERK signaling with viability, loss of the Fv2E-PERK transgene was observed in all cells that were able to proliferate in the presence of AP20187 (Fig. S3).