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We next evaluated whether Oxtr deletion impacted mature DGCs.
We also evaluated whether inhibition of DHODH has anti-proliferative effects in vitro.
We first evaluated whether progressively lower sampling effort (i.e., uncounted trees) could explain the trend.
We next evaluated whether NETs formation was pathogenic in LPS-mediated ALI.
We evaluated whether oxidative stress lead to an increase in cardiomyocyte-committed progenitor cells.
We also evaluated whether Gal-3 influenced C. neoformans growth and stability of EVs.
We evaluated whether these enzymes are expressed in O. viverrini.
We further evaluated whether there are changes in other cognitive behaviors due to ablation of GluN2C subunit.
We further evaluated whether body mass index (BMI) may mediate the association between weight loss intent and use of LCSs.
Also, we evaluated whether the parameter values changed depending on the surgical technique using the one-way ANOVA method.
We then evaluated whether exosomes derived from ARMS cells would influence proliferation of ERMS cells, and vice versa.
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