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Etiological hypotheses for FGID include low-level or transient inflammation of part of the GI tract [ 13, 14], altered intestinal motility [ 15] or visceral hypersensitivity [ 16], each of which suggests the need for different therapeutic approaches in specific patient groups.
Several etiological hypotheses related to intrinsic (sociodemographic, anthropometric, hormonal, immunological, genetic) and exogenous (ionizing radiation, electromagnetic fields, diet, infections, pesticides, drugs) risk factors have led to analytical epidemiological studies to establish relationships with CNS tumors.
Proper storage of such samples is required to allow epidemiological studies to be performed for the testing of different etiological hypotheses or, in general, to develop any bio-sanitary study to improve conservation strategies within the natural habitat.
A wide array of etiological hypotheses has been suggested to underlie depression.
Several underlying etiological hypotheses can be generated, to be tested in other confirmatory studies.
This distinction is likely to argue in favor of different etiological hypotheses.
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The availability of many etiological hypothesis for Alzheimer's disease raise the possibility of treatments aiming at slowing down disease progression.
These patterns were consistent with the genetic susceptibility etiological hypothesis [ 1, 2] related to HL.
The etiological hypothesis is that catecholamine release leads to these cardiopulmonary abnormalities.
This theory alone does not explain the pathophysiology of this process, and the explanation requires a multifactorial etiological hypothesis.
Animal experiments may help to understand the biology of these non-motor symptoms and test novel etiological hypothesis.
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