Sentence examples for At prm from inspiring English sources

During nighttime, it is observed that the H-component power spectra are larger at CB than at PRM, with the power difference decreasing toward longer periods.

During daytime, it is observed that the H spectra at CB are larger than at PRM for periods shorter than 80 s.

This can be explained by the amplitude enhancement at CB due to the crustal conductor and by the amplitude damping at PRM in the Pc3 pulsation range due to the Cowling conductivity of the EEJ (Sarma and Sastry 1995).

In periods longer than 80 s, amplification of the H-component by the EEJ prevails and the spectra are higher at PRM than at CB. Fig. 6 Comparison of the power spectra of the horizontal components of the magnetic field at two stations during the 1994 experiment.

As expected, the daily range (maximum value of H in relation to the base level at night) is observed around noon (1540 UT, equivalent to 1140 LT) and increases toward the dip equator (133 nT at PRM, 116 120 nT at the pair ARI and VIL, 92 93 nT at the pair COL and POV and 50 nT at CB). Fig. 2 Variations of the magnetic field H-component at stations near the magnetic equator on October 15 , 1994

At the C-prM site, lineage 2 strains are cleaved slightly more efficiently than lineage 1 strains; at prM-E, the reverse is true.

As reported previously, sequencing the prM-E genes of TM171-03 (V1 passage) identified nonsynonymous mutations encoding substitutions at prM-141 Ile→Thr and E156 Ser→Pro (2 ).

bAverage survival time ± SD was calculated for To confirm that the mouse virulence differences between the plaque-purified variants could be primarily attributed to the mutation at E-156, regions that encoded the additional consensus amino acid mutations at prM-141, NS4B-245, and NS5-898 were sequenced.

Given the greater degree of attenuation of neuroinvasiveness and neurovirulence observed for the nonglycosylated TM171-03-pp1 and -pp2 variants described here, we hypothesize that one or more of the other mutations (at prM-141, NS4B-245, or NS5-898) also contributed to the phenotype, but this hypothesis remains to be determined experimentally.

Table 1 shows the calculated hemolysis indices for the ULFED (at 4000 PRM) and the control device.

DO is nil (0 mg/l) at site S1 during PRM, attributed chiefly by the high stagnancy of the water source due to lack of sufficient flow.