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Analysis demonstrated that IgG1 was the dominant serum antibody isotype produced (Figure 4B).
Subsequent immunohistochemical analysis demonstrated that i.t.
ChIP analysis demonstrated that both Rad51 and Rad52 are recruited to Flex1 (Fig. 5c).
Gene expression analysis demonstrated that EFEMP1, an extracellular matrix protein, is associated with TMZ-resistant phenotype.
In vitro analysis demonstrated that JAK2V617F is a constitutively active tyrosine kinase.
Zeta potential analysis demonstrated that the microspheres were negatively charged.
Histological analysis demonstrated that NCSC transplantation promoted axonal myelination.
This analysis demonstrated that TM0415 utilizes PPi but neither ATP nor ADP to generate myo-inositol monophosphate (Fig. 2a).
Kaplan-Meier analysis demonstrated that vasodilator responders had significantly improved survival (P <.01).
CONCLUSION Longitudinal MRI analysis demonstrated that NeoCart-based repair tissue is durable and evolves over time.
Pathway analysis demonstrated that mutations in MAP2K4, a MAP3K1 substrate, produced similar perturbations as MAP3K1 loss.
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