We genotyped 1449 individuals from 57 populations and measured fitness-related traits in 10 populations of Pulsatilla vulgaris, a flagship species of calcareous grasslands in Europe.
It was therefore conducted a molecular analysis on individuals belonging to 10 populations in order to assess levels and distribution of genetic variation within and between populations.
We collected single-cell chromatin accessibility profiles across 10 populations of immunophenotypically defined human hematopoietic cell types and constructed a chromatin accessibility landscape of human hematopoiesis to characterize differentiation trajectories.
Among the 11 putative resistant populations, 10 populations evolved resistance to nicosulfuron, 6 populations were resistant to imazapic, and 4 populations evolved resistance to fomesafen, whereas none of the populations were resistant to bentazon.
We constructed LD maps for the 10 populations on autosomes.
Hence 74 colonies representing 10 populations were retained.
We collected seed from 20 25 randomly-chosen individuals from the 10 populations of study.
Eighty-six colonies from 13 populations for P. phylax and 75 colonies from 10 populations of C. mckeyi were entirely collected.
We then calculated the average MAF difference for pair-wise comparisons of human (n = 20) and CD-1 mouse (n = 10) populations.
A total of 188 individuals from 10 populations were genotyped with two sets of presumed neutral nuclear SNP loci previously described [15].
In the first scenario (PDN1), P = 10 populations each of a constant (diploid) size N = 500 were simulated from a common ancestral population with a star shaped phylogeny.
