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However, the mRNA levels of plastidial pyruvate kinase 3 (TDF #35_1) and aconitate hydratase 1 (TDF #33_2) genes were reduced in B-toxic C. sinensis leaves (Table 2).
The up-regulation of PPase 1 (TDF #118_1) in B-toxic C. sinensis leaves (Table 2) might be an adaptive response to B-toxicity.
Thus, the B-tolerance of C. grandis leaves might be down-regulated due to decreased transcript level of genes encoding 14-3-3 14-3-3 14-3-3#108_2) and heterotrimeric GTproteinng proTDFn subunit beta 108_2F #200_1) (Tande 2).
Thus, genes related to TFs might be affected in Mn-toxicity C. grandis leaves due to altered expression level of MAPK 1 (TDF #089-1) gene (Additional file 2).
The up-regulation of lecithin-cholesterol acyltransferase-like 1 (TDF #153-2, Additional file 2) in Mn-toxicity C. grandis leaves might enhance the leaf concentration of sterol ester, thus preventing leaf senescence.
The up-regulation of genes encoding MND1-interacting protein 1 (TDF #148-1) and ubiquitin-conjugating enzyme E2 10 (TDF #240-1) in Mn-toxicity C. grandis leaves (Additional file 2) might be an adaptive response to Mn-toxicity.
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