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This result clearly indicates that the molecules of 1 stimulate the proliferation of mES cells.
Increased plasma amino acid and glucose levels after a mixed meal and sulfonylureas normally stimulate β-cell secretion; increased plasma amino acid and perhaps glucose (2) levels after a mixed meal and sulfonylureas (1) stimulate α-cell secretion.
Studies have demonstrated that physical activity and fitness: 1) stimulate neural development, increasing the density of neuronal synapses [ 39], 2) increase levels of norepinephrine and endorphins, which are important to reduce stress and improve mood [ 40]; and 3) might increase the vasculature in the cerebral cortex [ 41].
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How does Ca2+ stimulate release so rapidly and precisely?
LPS and IL-1 stimulate IκB kinase activity, resulting in the subsequent IκB phosphorylation and ubiquitinylation.
Consistent with this general pathway we show that both LPS and IL-1 stimulate NF-κB transcriptional activity as well as result in IκB degradation.
The inhibitory effect of a LIFR antagonist on osteoblast differentiation is consistent with reports that the LIFR ligands LIF and CT-1 stimulate osteoblast differentiation and bone formation [23], [42] and that OSM, acting through LIFR in mice, stimulates bone formation [43].
These findings suggest that RASSF1A and RASSF5 stimulate MST signaling.
Both TNF-α and IL-1β stimulate NFκB activation.
Hypoxia, a common feature of the inflamed synovial environment, and IL-1 stimulate VEGF expression in synovial fibroblasts [ 109].
During the first step, hypoxia and HIF-1 stimulate VEGF and their receptors directly, and the cascade of new vessel creation begins.
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