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sg 1 MAb 3/1 negative, and all other structures colonized by L. pneumophila sg 2 14.
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In the process, we have identified the epitope recognized by anti-NY-ESO-1 mAb E978 (79–87, GARGPESRL).
The combination of antigen-specific aAPC-based activation and anti-PD-1 mAb checkpoint blockade induced the greatest IFN-γ secretion by CD8+ T cells in vitro.
Pretreatment with anti-LOX-1 mAb, Vitamin C, apocynin or catalase significantly reduced ROS production and prevented ox-LDL-induced apoptosis, while indomethacin or allopurinol had no effect.
LAT-1 was stained by using an anti-LAT-1 mAb (rabbit IgG, synthetic peptide, Abcam).
Addition of anti-ICAM-1 mAb to the coculture of T and B cells inhibited T cell proliferation by approximately 20%.
Nivolumab is an anti-PD-1 mAb that provides a durable response in various advanced malignancies [2, 3].
The induction of neutropenia, via depletion with Gr-1 mAb or myeloid-specific ablation of Mcl-1, elicits G-CSF via an IL-1RI-independent pathway, stimulating granulopoietic responses indistinguishable from those induced by adjuvant.
Anti-human-VAP-1 mAb 1B2 was labeled with 123I using the chloramine T method and evaluated in animal models of skin inflammation and aseptic arthritis.
Anti-F4/80 (A3-1) mAb was purchased from Serotec.
We first grafted particles to saturation with an anti-LFA-1 mAb (CD18).
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