Exact(5)
In experiment 1, lactating Holstein cows (n = 112) were fitted with an accelerometer system and were treated with GnRH followed 7 d later by PGF2α to synchronize estrus.
In Experiment 1, lactating beef cows (n=140) and pubertal heifers (n=40) were randomly allocated to three groups to receive 100 μg gonadorelin (GnRH), 5 mg estradiol-17β and 100 mg progesterone (E+P) in canola oil or no treatment (control) on day 0. All cattle were observed for estrus every 12 h from 36 to 96 h after PGF.
The distribution of the pathology of these cases was as follows: intraductal papilloma (n = 6), fibrocystic change (n = 5), atypical ductal hyperplasia (n = 4), fibrous mastopathy (n = 2), papillary lesion (n = 1), sclerosing adenosis (n = 1), lactating adenosis (n = 1), atypical cell (n = 1), chronic inflammation (n = 1), and other (n = 1) (Table 4).
All 3 cats were female (2 pregnant, 1 lactating); prevalence was significantly higher in pregnant or lactating females (3/12) than other intact females (0/24) (p = 0.031 by Fisher exact test).
Whole mounts of day 1 lactating (D1L) mammary glands from multiparous Ndst1f/fMMTVCre+ females revealed dramatically retarded lobuloalveolar expansion compared to lactating glands from wildtype animals (compare Figs. 4A and 4B), whereas whole mounts of multiparous non-pregnant Ndst1f/fMMTVCre+ females looked normal (data not shown).
Similar(55)
The experimental design was a replicated 4 × 4 Latin square with 8 lactating dairy cows.
A purposive sample of 40 lactating Holstein cows was selected for detailed observation on each farm.
The experiment was designed as a replicated 3 × 3 Latin square using 6 lactating dairy cows with ruminal cannulas.
Thus, 20 women- 12 lactating mothers and 8 currently pregnant women were selected for the study.
Figure 1 Lactate at the cellular level.
Table 1 Lactate levels according to outcome.
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