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Open image in new window Fig. 1 DRS spectra of ZnS and ZnS-Ag nanoballs Open image in new window Fig. 2 Tauc Plot of Ag doped ZnS.
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Table 3 DRs of the proposed method (Prop).
These results showed the inhibitory potential of IPAD on HSV wild types and HSV-1 DRs and suggested that IPAD could be used in combination with ACV for treatment of HSV-1 DRs infections.
In contrast, the IC50 and SI values of ACV against HSV wild types and HSV-1 DRs were markedly different.
In the present study, anti-viral activity of IPAD against HSV wild types and HSV-1 DRs was evaluated.
IPAD was spectacularly effective in inhibiting HSV-1 DRs, presumably involving a mode of action different from ACV or phosphonoacetate.
The IC50 values for ACV against HSV wild types were less than 1.0 μM, whereas those against HSV-1 DRs were more than 160 μM (Table 1).
Interestingly, the IC50 as well as SI values of IPAD against HSV wild types and HSV-1 DRs were not different (Table 1).
A non-cytotoxic concentration of IPAD (20.50 μM) completely inhibited CPE formation induced by HSV wild types and HSV-1 DRs after viral entry into the cells.
In the present study, the potential synergistic effects between IPAD and acyclovir were also observed against both HSV wild types and HSV-1 DRs (Table 2).
Base on the inhibitory effect of IPAD on viral DNA and protein synthesis of HSV-1 DRs, this study suggested that IPAD and ACV have different targets.
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