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1 BRAIN ON FIRE by Susannah Cahalan.
13613_2011_59_MOESM1_ESM.DOC Additional file 1: Brain Death Survey.
Fig. 1 Brain magnetic resonance imaging T2/FLAIR image.
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Three patients were excluded because of the possibility of secondary NDPH (2 patients: benign intracranial hypertension, 1: brain tuberculoma).
Data analysis examined the effects of cortisol administration on 1) brain activation during encoding, and 2) subsequent free recall for words.
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Further studies revealed that PME-1 brain tissue also possessed significantly reduced PP2A activity with phosphopeptide substrates and diminished quantities of PP2A holoenzyme complexes.
These data established that the residual PrPC that persisted in pronase E-digested normal CD-1 brain homogenate was completely eliminated from the NaPTA precipitated pellet fraction (Figures 4 and 5).
Single photon emission computerized tomography with [ 99m Tc]-TRODAT-1 brain scans will be performed to evaluate dopamine transporters.
In normal CD-1 brain homogenate, PrPC level is around ∼20% of total PrP present in untreated RML brain homogenate.
Paraffin-embedded, formalin-fixed brain tissues from 22 patients were obtained from the Manhattan HIV-1 Brain Bank, National NeuroAIDS Tissue Consortium (37).
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