However, the IFIH1 variants are protective, which will tend to increase IBD in control/control pairs, while our IBD test looks for increased IBD in case/case pairs.
Whether AAs represent an important population with IBD in the US remains unclear since few studies have investigated IBD in communities with a majority representation of AA patients.
Classical assumptions about the prevalence and course of IBD in AAs may underlie the failure to suspect, diagnose, and treat IBD in this population.
To determine the characteristics of inflammatory bowel disease (IBD) in young patients.
It is reported that PSC is strongly associated with inflammatory bowel disease (IBD); in fact, its rate of incidence among patients with IBD is 75% [1].
The article presents a population-based registry designed to estimate incidence and prevalence of inflammatory bowel disease (IBD) in the area of Forlì (north-eastern Italy).
To assess the contribution of the 113 G→A missense mutation within the discs, large homolog 5 (DLgenegene in childhood-onset inflammatory bowel disease (IBD) in Scotland.
We wished to determine whether infection with either H. bilis or H. hepaticus would accelerate the development of inflammatory bowel disease (IBD) in mdr1a−/− mice.
To determine the prevalence of a family history (FMH) of inflammatory bowel disease (IBD) in patients with idiopathic ocular inflammation (OI).
The aim of this study was to assess characteristics and predictive factors of complementary and alternative medicine (CAM) use for patients with inflammatory bowel disease (IBD) in Korea.
Exacerbated IBD in CCR9−/− mice further correlated with upregulation of IFNγ, IL-17A and IL-17F.
