Exact(60)
The presence of APL in both parental and HeLa-APL-resistant cell systems after 1 h exposure was confirmed, as demonstrated by the detection of the different ionised forms of APL, that is, ∼1110 for APL+H, ∼1132 for APL+Na and ∼1148 for APL+K.. Consistently, APL-dmac was also detected in both cell lines.
Thirty-three children with APL were included; 78.8% were treated with APL -specific protocols.
Co-immunization with each antagonistic APL markedly prevented the development of arthritis, especially APL 13 (G332V).
Older APL patients seem to be as sensitive to specific APL therapy as younger individuals.
In the presence of UFH or LMWH, however, complement activation was completely inhibited (C3adesArg [ng/ml]: aPL, 680 ± 30; UFH + aPL, 120 ± 20*; LMWH + aPL, 105 ± 18*; % C3-positive BeWo cells by FACS: aPL, 31 ± 11; aPL + UFH, 9 ± 5*; LMWH + aPL, 5 ± 3; * P < 0.01 versus aPL).
Since APL appeared to be unable to induce apoptosis in HeLa-APL-resistant cells, we studied the molecular effects of APL in both cell systems.
Patients with APL were excluded.
Huge relief at APL: On to Pluto!
NOL is the parent company of APL.
The APL team decided to check that second scenario straightaway.
Many people at APL have been with New Horizons since the launch in 2006.
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Since I tried Ludwig back in 2017, I have been constantly using it in both editing and translation. Ever since, I suggest it to my translators at ProSciEditing.

Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com